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Histological Evaluation of the Temporomandibular Joint after Producing Artificial Traumatic Occlusion in Dogs | ||
Journal of Dentistry | ||
مقاله 1، دوره 11، Supplement Winter 2010، آذر 2010، صفحه 1-7 اصل مقاله (114.81 K) | ||
نوع مقاله: Original Article | ||
شناسه دیجیتال (DOI): 10.30476/dentjods.2019.43590 | ||
نویسندگان | ||
A. Madani1؛ N. Mohtasham2؛ M. Jazayeri3؛ F. Shahabian* 1؛ M. Shakeri4 | ||
1Dept. of Prosthodontics, School of Dentistry, Mashhad University of Medical Sciences, Mashhad, Iran | ||
2Dept. of Oral and Maxillofacial Pathology, School of Dentistry, Mashhad University of Medical Sciences, Mashhad, Iran | ||
3Dept. of Oral Medicine, School of Dentistry, Hamadan University of Medical Sciences, Hamadan, Iran | ||
4Public Health Department of Ghaem Hospital, Mashhad University of Medical Sciences | ||
چکیده | ||
Statement of Problem: Traumatic occlusion can have an adverse effect on the temporomandibular joint tissue.Purpose: This study aims to determine the histological alteration in dog-s’ temporomandibular joints due to production of artificial traumatic occlusion.Materials and Methods: Twenty dogs were divided into a control group of four and two equal experimental groups. Unilateral occlusal interferences were created on the mandibular right first molars in the experimental groups. We established two experimental periods: one week and 45 days. After vital perfusion, the TMJs were separated for preparation, pigmentation, and examination by a pathologist. Descriptive statistics, Chi-square and Fisher's exact test were used to analyze the data. P.value less than 0.05 was considered as significant.Results: The findings showed no inflammatory changes in the TMJ. Increased fibrosis was found in 56.30% and vascularization in 28.12% of the disks. Fibrotic changes were detected in 46.87% and vascularization in 28.12% of the condyles. The glenoid fossas suffered from fibrosis in 15.62% of the cases.Conclusion: Traumatic occlusion leads to histopathological alterations including an increase in the fibrosis tissue and vascularization in the condyle and disk.KEY WORDS: Dental occlusion; Traumatic; Histology; Temporomandibular joint | ||
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